FGFR2 point mutations in 466 endometrioid endometrial tumors: Relationship with MSI, KRAS, PIK3CA, CTNNB1 mutations and clinicopathological features

Authors

Sara A. Byron, Translational Genomics Research InstituteFollow
Michael Gartside, Translational Genomics Research Institute
Matthew A. Powell, Washington University School of Medicine in St. Louis
Candice L. Wellens, Translational Genomics Research Institute
Feng Gao, Washington University School of Medicine in St. LouisFollow
David G. Mutch, Washington University School of Medicine in St. Louis
Paul J. Goodfellow, Washington University School of Medicine in St. LouisFollow
Pamela M. Pollock, Translational Genomics Research Institute

Journal

PLoS ONE

Publication Date

2012

Volume

7

Issue

2

Inclusive Pages

1-9

Document Type

Open Access Publication

DOI

10.1371/journal.pone.0030801

Rights and Permissions

Byron SA, Gartside M, Powell MA, Wellens CL, Gao F, et al. (2012) FGFR2 Point Mutations in 466 Endometrioid Endometrial Tumors: Relationship with MSI, KRAS, PIK3CA, CTNNB1 Mutations and Clinicopathological Features. PLoS ONE 7(2): e30801. doi:10.1371/journal.pone.0030801

Recommended Citation

Byron, Sara A.; Gartside, Michael; Powell, Matthew A.; Wellens, Candice L.; Gao, Feng; Mutch, David G.; Goodfellow, Paul J.; and Pollock, Pamela M., "FGFR2 point mutations in 466 endometrioid endometrial tumors: Relationship with MSI, KRAS, PIK3CA, CTNNB1 mutations and clinicopathological features." PLoS ONE. 7, 2. 1-9. (2012).
https://digitalcommons.wustl.edu/open_access_pubs/1009