Arhgap24 inactivates Rac1 in mouse podocytes, and a mutant form is associated with familial focal segmental glomerulosclerosis

Authors

Shreeram Akilesh, Barnes-Jewish HospitalFollow
Hani Suleiman, Washington University School of Medicine in St. Louis
Christine Stander, Washington University School of Medicine in St. Louis
Haiyang Yu, Washington University School of Medicine in St. Louis
Peter Lavin, Duke University Medical Center
Rasheed Gbadegesin, Duke University Medical Center
Corinne Antignac, Hopital Necker-Enfants-Malades Tour LavoisierFollow
Martin Pollack, Harvard Medical School
Jeffrey B. Kopp, National Institute for Health
Michelle P. Winn, Duke University Medical Center
Andrey S. Shaw, Washington University School of Medicine in St. Louis

Journal

The Journal of Clinical Investigation

Publication Date

2011

Volume

121

Issue

10

Inclusive Pages

4127-4137

Document Type

Open Access Publication

DOI

10.1172/JCI46458

Rights and Permissions

Arhgap24 inactivates Rac1 in mouse podocytes, and a mutant form is associated with familial focal segmental glomerulosclerosis Shreeram Akilesh, Hani Suleiman, Haiyang Yu, M. Christine Stander, Peter Lavin, Rasheed Gbadegesin, Corinne Antignac, Martin Pollak, Jeffrey B. Kopp, Michelle P. Winn, Andrey S. Shaw Published in Volume 121, Issue 10 J Clin Invest. 2011; 121(10):4127–4137 doi:10.1172/JCI46458

Recommended Citation

Akilesh, Shreeram; Suleiman, Hani; Stander, Christine; Yu, Haiyang; Lavin, Peter; Gbadegesin, Rasheed; Antignac, Corinne; Pollack, Martin; Kopp, Jeffrey B.; Winn, Michelle P.; and Shaw, Andrey S., "Arhgap24 inactivates Rac1 in mouse podocytes, and a mutant form is associated with familial focal segmental glomerulosclerosis." The Journal of Clinical Investigation. 121, 10. 4127-4137. (2011).
https://digitalcommons.wustl.edu/open_access_pubs/1691