Kir6.2 variant E23K increases ATP-sensitive K+ channel activity and is associated with impaired insulin release and enhanced insulin sensitivity in adults with normal glucose tolerance

Authors

Dennis T. Villareal, Washington University School of Medicine in St. Louis
Joseph C. Koster, Washington University School of Medicine in St. LouisFollow
Heather Robertson, Washington University School of Medicine in St. Louis
Alejandro Akrouh, Washington University School of Medicine in St. Louis
Kazuaki Miyake, University of Chicago
Graeme I. Bell, University of Chicago
Bruce W. Patterson, Washington University School of Medicine in St. Louis
Colin G. Nichols, Washington University School of Medicine in St. Louis
Kenneth S. Polonsky, Washington University School of Medicine in St. Louis

Areas of Expertise

Dennis T. Villareal, 6680 Geriatrics; Colin G. Nichols, 3360 Molecular Medicine; Kenneth S. Polonsky, 6630 Diabetes

Journal

Diabetes

Volume

58

Issue

8

Document Type

Article

Pages

1869-1878

Publication Date

2009

Recommended Citation

Villareal, Dennis T.; Koster, Joseph C.; Robertson, Heather; Akrouh, Alejandro; Miyake, Kazuaki; Bell, Graeme I.; Patterson, Bruce W.; Nichols, Colin G.; and Polonsky, Kenneth S., "Kir6.2 variant E23K increases ATP-sensitive K+ channel activity and is associated with impaired insulin release and enhanced insulin sensitivity in adults with normal glucose tolerance". Diabetes, 1869-1878. 2009. Paper 38.
https://digitalcommons.wustl.edu/icts_facpubs/38