Kir6.1- and SUR2-dependent KATP over-activity disrupts intestinal motility in murine models of Cantu Syndrome

Authors

Nathaniel W York, Washington University School of Medicine in St. Louis
Helen Parker, Washington University School of Medicine in St. Louis
Zili Xie, Washington University School of Medicine in St. Louis
David Tyus, Washington University School of Medicine in St. Louis
Maham A Waheed, Washington University School of Medicine in St. Louis
Zihan Yan, Washington University School of Medicine in St. Louis
Dorothy K Grange, Washington University School of Medicine in St. Louis
Maria S Remedi, Washington University School of Medicine in St. Louis
Sarah K England, Washington University School of Medicine in St. Louis
Hongzhen Hu, Washington University School of Medicine in St. Louis
Colin G Nichols, Washington University School of Medicine in St. Louis

Journal

JCI Insight

Publication Date

11-10-2020

Volume

5

Issue

23

First Page

e141443

Document Type

Open Access Publication

DOI

10.1172/jci.insight.141443

Rights and Permissions

York NW, Parker H, Xie Z, Tyus D, Waheed MA, Yan Z, Grange DK, Remedi MS, England SK, Hu H, Nichols CG. Kir6.1- and SUR2-dependent KATP over-activity disrupts intestinal motility in murine models of Cantu Syndrome. JCI Insight. 2020 Nov 10;5(23):e141443. doi: 10.1172/jci.insight.141443. © 2020, York et al. This is an open access article published under the terms of the Creative Commons Attribution 4.0 International License.

Recommended Citation

York, Nathaniel W; Parker, Helen; Xie, Zili; Tyus, David; Waheed, Maham A; Yan, Zihan; Grange, Dorothy K; Remedi, Maria S; England, Sarah K; Hu, Hongzhen; and Nichols, Colin G, "Kir6.1- and SUR2-dependent KATP over-activity disrupts intestinal motility in murine models of Cantu Syndrome." JCI Insight. 5, 23. e141443 (2020).
https://digitalcommons.wustl.edu/open_access_pubs/10070