Journal
Proceedings of the National Academy of Sciences of the United States of America
Publication Date
2021
Volume
118
Issue
14
First Page
e2014492118
Document Type
Open Access Publication
DOI
10.1073/pnas.2014492118
Rights and Permissions
Manouchehri N, Hussain RZ, Cravens PD, Esaulova E, Artyomov MN, Edelson BT, Wu GF, Cross AH, Doelger R, Loof N, Eagar TN, Forsthuber TG, Calvier L, Herz J, Stüve O. CD11c+CD88+CD317+ myeloid cells are critical mediators of persistent CNS autoimmunity. Proc Natl Acad Sci U S A. 2021 Apr 6;118(14):e2014492118. doi: 10.1073/pnas.2014492118 Copyright © 2021 the Author(s). Published by PNAS. This open access article is distributed under Creative Commons Attribution-NonCommercial-NoDerivatives License 4.0 (CC BY-NC-ND).
Recommended Citation
Manouchehri, Navid; Hussain, Rehana Z.; Cravens, Petra D.; Esaulova, Ekaterina; Artyomov, Maxim N.; Edelson, Brian T.; Wu, Gregory F.; Cross, Anne H.; Doelger, Richard; Loof, Nicolas; Eagar, Todd N.; Forsthuber, Thomas G.; Calvier, Laurent; Herz, Joachim; and Stüve, Olaf, "CD11c+ CD88+ CD317+ myeloid cells are critical mediators of persistent CNS autoimmunity." Proceedings of the National Academy of Sciences of the United States of America. 118, 14. e2014492118 (2021).
https://digitalcommons.wustl.edu/open_access_pubs/10229