Aging-associated deficit in CCR7 is linked to worsened glymphatic function, cognition, neuroinflammation, and β-amyloid pathology

Authors

Sandro Da Mesquita, University of Virginia
Jasmin Herz, Washington University School of Medicine in St. Louis
Morgan Wall, University of Virginia
Taitea Dykstra, Washington University School of Medicine in St. Louis
Kalil Alves de Lima, Washington University School of Medicine in St. Louis
Geoffrey T Norris
Nisha Dabhi, University of Virginia
Tatiana Kennedy, University of Virginia
Wendy Baker, University of Virginia
Jonathan Kipnis, Washington University School of Medicine in St. Louis

Journal

Science Advances

Publication Date

2021

Volume

7

Issue

21

Document Type

Open Access Publication

DOI

10.1126/sciadv.abe4601

Rights and Permissions

Da Mesquita S, Herz J, Wall M, etCopyright © 2021 The Authors, some rights reserved; exclusive licensee American Association for the Advancement of Science. No claim to original U.S. Government Works. Distributed under a Creative Commons Attribution NonCommercial License 4.0 (CC BY-NC). al. Aging-associated deficit in CCR7 is linked to worsened glymphatic function, cognition, neuroinflammation, and β-amyloid pathology. Sci Adv. 2021;7(21):eabe4601. Published 2021 May 21. doi:10.1126/sciadv.abe4601.

Recommended Citation

Da Mesquita, Sandro; Herz, Jasmin; Wall, Morgan; Dykstra, Taitea; de Lima, Kalil Alves; Norris, Geoffrey T; Dabhi, Nisha; Kennedy, Tatiana; Baker, Wendy; and Kipnis, Jonathan, "Aging-associated deficit in CCR7 is linked to worsened glymphatic function, cognition, neuroinflammation, and β-amyloid pathology." Science Advances. 7, 21. (2021).
https://digitalcommons.wustl.edu/open_access_pubs/10427