Myocardial afterload is a key biomechanical regulator of atrioventricular myocyte differentiation in zebrafish

Authors

Neha Ahuja, Colorado State University
Paige Ostwald, Colorado State University
Alex Gendernalik, Colorado State University
Elena Guzzolino, Meyer's Children's Hospital
Letizia Pitto, National Council of Research
David Bark, Washington University School of Medicine in St. Louis
Deborah M Garrity, Colorado State University

Journal

Journal of Cardiovascular Development and Disease

Publication Date

1-12-2022

Volume

9

Issue

1

First Page

22

Document Type

Open Access Publication

DOI

10.3390/jcdd9010022

Rights and Permissions

Ahuja N, Ostwald P, Gendernalik A, Guzzolino E, Pitto L, Bark D, Garrity DM. Myocardial Afterload Is a Key Biomechanical Regulator of Atrioventricular Myocyte Differentiation in Zebrafish. J Cardiovasc Dev Dis. 2022 Jan 12;9(1):22. doi: 10.3390/jcdd9010022. © 2022 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

Recommended Citation

Ahuja, Neha; Ostwald, Paige; Gendernalik, Alex; Guzzolino, Elena; Pitto, Letizia; Bark, David; and Garrity, Deborah M, "Myocardial afterload is a key biomechanical regulator of atrioventricular myocyte differentiation in zebrafish." Journal of Cardiovascular Development and Disease. 9, 1. 22 (2022).
https://digitalcommons.wustl.edu/open_access_pubs/11241