Journal
Epigenetics
Publication Date
1-1-2022
Volume
17
Issue
2
First Page
220
Last Page
238
Document Type
Open Access Publication
DOI
10.1080/15592294.2021.1954780
Rights and Permissions
Maurya S, Yang W, Tamai M, Zhang Q, Erdmann-Gilmore P, Bystry A, Martins Rodrigues F, Valentine MC, Wong WH, Townsend R, Druley TE. Loss of KMT2C reprograms the epigenomic landscape in hPSCs resulting in NODAL overexpression and a failure of hemogenic endothelium specification. Epigenetics. 2022 Jan-Feb;17(2):220-238. doi.org/10.1080/15592294.2021.1954780 This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivatives License (http://creativecommons.org/licenses/by-nc-nd/4.0/), which permits non-commercial re-use, distribution, and reproduction in any medium, provided the original work is properly cited, and is not altered, transformed, or built upon in any way.
Recommended Citation
Maurya, Shailendra; Yang, Wei; Tamai, Minori; Zhang, Qiang; Erdmann-Gilmore, Petra; Bystry, Amelia; Martins Rodrigues, Fernanda; Valentine, Mark C; Wong, Wing H; Townsend, Reid; and Druley, Todd E, "Loss of KMT2C reprograms the epigenomic landscape in hPSCs resulting in NODAL overexpression and a failure of hemogenic endothelium specification." Epigenetics. 17, 2. 220 - 238. (2022).
https://digitalcommons.wustl.edu/open_access_pubs/11525
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