Loss of Par-1a/MARK3/C-TAK1 kinase leads to reduced adiposity, resistance to hepatic steatosis, and defective gluconeogenesis

Authors

Jochen K. Lennerz, Washington University School of Medicine in St. LouisFollow
Jonathan B. Hurov, Washington University School of Medicine in St. LouisFollow
Lynn S. White, Washington University School of Medicine in St. Louis
Katherine T. Lewandowski, Washington University School of Medicine in St. Louis
Julie L. Prior, Washington University School of Medicine in St. Louis
G. James Planer, Washington University School of Medicine in St. Louis
Robert W. Gereau IV, Washington University School of Medicine in St. LouisFollow
David Piwnica-Worms, Washington University School of Medicine in St. Louis
Robert E. Schmidt, Washington University School of Medicine in St. Louis
Helen Piwnica-Worms, Washington University School of Medicine in St. Louis

Journal

Molecular and Cellular Biology

Publication Date

2010

Volume

30

Issue

21

Inclusive Pages

5043-5056

Document Type

Open Access Publication

DOI

10.1128/MCB.01472-09

Rights and Permissions

Loss of Par-1a/MARK3/C-TAK1 kinase leads to reduced adiposity, resistance to hepatic steatosis, and defective gluconeogenesis, Jochen K. Lennerz , Jonathan B. Hurov, Lynn S. White, Katherine T. Lewandowski, Julie L. Prior, G. James Planer, Robert W. Gereau IV, David Piwnica-Worms, Robert E. Schmidt, and Helen Piwnica-Worms Mol. Cell. Biol. November 1, 2010 30:21 5043-5056; published ahead of print 23 August 2010, doi:10.1128/MCB.01472-09

Recommended Citation

Lennerz, Jochen K.; Hurov, Jonathan B.; White, Lynn S.; Lewandowski, Katherine T.; Prior, Julie L.; Planer, G. James; Gereau, Robert W. IV; Piwnica-Worms, David; Schmidt, Robert E.; and Piwnica-Worms, Helen, "Loss of Par-1a/MARK3/C-TAK1 kinase leads to reduced adiposity, resistance to hepatic steatosis, and defective gluconeogenesis." Molecular and Cellular Biology. 30, 21. 5043-5056. (2010).
https://digitalcommons.wustl.edu/open_access_pubs/2744