Early neural and vascular dysfunctions in diabetic rats are largely sequelae of increased sorbitol oxidation

Authors

Yasudo Ido, Boston University School of Medicine
Jens R. Nyengaard, University of Aarhus, Denmark
Kathy Chang, Washington University School of Medicine in St. LouisFollow
Ronald G. Tilton, University of Texas Medical Branch
Charles Kilo, Washington University School of Medicine in St. Louis
Banavara L. Mylari, Pfizer Global Research and Development
Peter J. Oates, Pfizer Global Research and Development
Joseph R. Williamson, Washington University School of Medicine in St. Louis

Journal

Antioxidants & Redox Signaling

Publication Date

2009

Volume

12

Issue

1

Inclusive Pages

39-51

Document Type

Open Access Publication

DOI

10.1089/ars.2009.2502

Rights and Permissions

Yasuo Ido, Jens R. Nyengaard, Kathy Chang, Ronald G. Tilton, Charles Kilo, Banavara L. Mylari, Peter J. Oates, and Joseph R. Williamson. Antioxidants & Redox Signaling. January 1, 2010, 12(1): 39-51. doi:10.1089/ars.2009.2502.

Recommended Citation

Ido, Yasudo; Nyengaard, Jens R.; Chang, Kathy; Tilton, Ronald G.; Kilo, Charles; Mylari, Banavara L.; Oates, Peter J.; and Williamson, Joseph R., "Early neural and vascular dysfunctions in diabetic rats are largely sequelae of increased sorbitol oxidation." Antioxidants & Redox Signaling. 12, 1. 39-51. (2009).
https://digitalcommons.wustl.edu/open_access_pubs/2831