Mitochondrial pyruvate carrier 2 hypomorphism in mice leads to defects in glucose-stimulated insulin secretion

Authors

Patrick A. Vigueira, Washington University School of Medicine in St. Louis
Kyle S. McCommis, Washington University School of Medicine in St. Louis
George G. Schweitzer, Washington University School of Medicine in St. Louis
Maria S. Remedi, Washington University School of Medicine in St. Louis
Kari T. Chambers, Washington University School of Medicine in St. Louis
Xiaorong Fu, University of Texas Southwestern Medical Center at Dallas
William G. McDonald, Metabolic Solutions Development Company
Serena L. Cole, Metabolic Solutions Development Company
Jerry R. Colca, Metabolic Solutions Development CompanyFollow
Rolf F. Kletzien, Metabolic Solutions Development CompanyFollow
Shawn C. Burgess, University of Texas Southwestern Medical Center at Dallas
Brian N. Finck, Washington University School of Medicine in St. LouisFollow

Journal

Cell Reports

Publication Date

2014

Volume

7

Inclusive Pages

1-12

Document Type

Open Access Publication

DOI

10.1016/j.celrep.2014.05.017

Rights and Permissions

Vigueira et al., Mitochondrial Pyruvate Carrier 2 Hypomorphism in Mice Leads to Defects in Glucose-Stimulated Insulin Secretion, Cell Reports (2014), http://dx.doi.org/10.1016/j.celrep.2014.05.017

Recommended Citation

Vigueira, Patrick A.; McCommis, Kyle S.; Schweitzer, George G.; Remedi, Maria S.; Chambers, Kari T.; Fu, Xiaorong; McDonald, William G.; Cole, Serena L.; Colca, Jerry R.; Kletzien, Rolf F.; Burgess, Shawn C.; and Finck, Brian N., "Mitochondrial pyruvate carrier 2 hypomorphism in mice leads to defects in glucose-stimulated insulin secretion." Cell Reports. 7, 1-12. (2014).
https://digitalcommons.wustl.edu/open_access_pubs/2970