Homozygous KSR1 deletion attenuates morbidity but does not prevent tumor development in a mouse model of RAS-driven pancreatic cancer

Authors

Elizabeth A. Germino, Washington University School of Medicine in St. Louis
Joseph P. Miller, Washington University School of Medicine in St. Louis
Lauri Diehl, Genentech, South San Francisco
Carter J. Swanson, Genentech, South San Francisco
Steffen Durinck, Genentech, South San Francisco
Zora Modrusan, Genentech, South San Francisco
Jeffrey H. Miner, Washington University School of Medicine in St. Louis
Andrey S. Shaw, Washington University School of Medicine in St. Louis

Journal

PLoS One

Publication Date

2018

Volume

13

Issue

3

Inclusive Pages

e0194998

Document Type

Open Access Publication

DOI

10.1371/journal.pone.0194998

Rights and Permissions

Germino EA, Miller JP, Diehl L, Swanson CJ, Durinck S, et al. (2018) Homozygous KSR1 deletion attenuates morbidity but does not prevent tumor development in a mouse model of RAS-driven pancreatic cancer. PLOS ONE 13(3): e0194998. https://doi.org/10.1371/journal.pone.0194998

Recommended Citation

Germino, Elizabeth A.; Miller, Joseph P.; Diehl, Lauri; Swanson, Carter J.; Durinck, Steffen; Modrusan, Zora; Miner, Jeffrey H.; and Shaw, Andrey S., "Homozygous KSR1 deletion attenuates morbidity but does not prevent tumor development in a mouse model of RAS-driven pancreatic cancer." PLoS One. 13, 3. e0194998. (2018).
https://digitalcommons.wustl.edu/open_access_pubs/6821