Crx-L253X mutation produces dominant photoreceptor defects in TVRM65 mice

Authors

Philip A. Ruzycki, Washington University School of Medicine in St. Louis
Courtney D. Linne, Washington University School of Medicine in St. Louis
Anne K. Hennig, Washington University School of Medicine in St. Louis
Shiming Chen, Washington University School of Medicine in St. Louis

Journal

Investigative Ophthalmology & Visual Science

Publication Date

2017

Volume

58

Issue

11

Inclusive Pages

4644–4653

Document Type

Open Access Publication

DOI

10.1167/iovs.17-22075

Rights and Permissions

Ruzycki PA, Linne CD, Henning AK, Chen S. Crx-L253X mutation produces dominant photoreceptor defects in TVRM65 mice. Invest Ophthalmol Vis Sci. 2017;58:4644–4653. DOI:10.1167/iovs.17-22075. This work is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License.

Recommended Citation

Ruzycki, Philip A.; Linne, Courtney D.; Hennig, Anne K.; and Chen, Shiming, "Crx-L253X mutation produces dominant photoreceptor defects in TVRM65 mice." Investigative Ophthalmology & Visual Science. 58, 11. 4644–4653. (2017).
https://digitalcommons.wustl.edu/open_access_pubs/7210