KATP channels are necessary for glucose-dependent increases in amyloid-β and Alzheimer's disease-related pathology

Authors

John Grizzanti, Wake Forest University
William R. Moritz, Washington University School of Medicine in St. Louis
Molly Stanley, Washington University School of Medicine in St. Louis
Emily E. Caesar, Washington University School of Medicine in St. Louis
Maria S. Remedi, Washington University School of Medicine in St. Louis
Celeste M. Karch, Washington University School of Medicine in St. Louis
Colin G. Nichols, Washington University School of Medicine in St. Louis
David M. Holtzman, Washington University School of Medicine in St. Louis
et al.

Journal

JCI Insight

Publication Date

5-2-2023

Volume

8

Issue

10

First Page

e162454

Document Type

Open Access Publication

DOI

10.1172/jci.insight.162454

Rights and Permissions

Grizzanti J, Moritz WR, Pait MC, Stanley M, Kaye SD, Carroll CM, Constantino NJ, Deitelzweig LJ, Snipes JA, Kellar D, Caesar EE, Pettit-Mee RJ, Day SM, Sens JP, Nicol NI, Dhillon J, Remedi MS, Kiraly DD, Karch CM, Nichols CG, Holtzman DM, Macauley SL. KATP channels are necessary for glucose-dependent increases in amyloid-β and Alzheimer's disease-related pathology. JCI Insight. 2023 May 2;8(10):e162454. doi: 10.1172/jci.insight.162454. © 2023, Grizzanti et al. This is an open access article published under the terms of the Creative Commons Attribution 4.0 International License.

Recommended Citation

Grizzanti, John; Moritz, William R.; Stanley, Molly; Caesar, Emily E.; Remedi, Maria S.; Karch, Celeste M.; Nichols, Colin G.; Holtzman, David M.; and et al., "KATP channels are necessary for glucose-dependent increases in amyloid-β and Alzheimer's disease-related pathology." JCI Insight. 8, 10. e162454 (2023).
https://digitalcommons.wustl.edu/oa_4/1729

Department

ICTS (Institute of Clinical and Translational Sciences)

Additional Links

Supplemental material is available for this article at publisher site.