Abrogation of MAP4K4 protein function causes congenital anomalies in humans and zebrafish

Authors

Victoria Patterson, Princeton University
Dorothy K Grange, Washington University School of Medicine in St. Louis
et al.

Journal

Science advances

Publication Date

4-28-2023

Volume

9

Issue

17

First Page

eade0631

Document Type

Open Access Publication

DOI

10.1126/sciadv.ade0631

Rights and Permissions

Patterson V, Ullah F, Bryant L, Griffin JN, Sidhu A, Saliganan S, Blaile M, Saenz MS, Smith R, Ellingwood S, Grange DK, Hu X, Mireguli M, Luo Y, Shen Y, Mulhern M, Zackai E, Ritter A, Izumi K, Hoefele J, Wagner M, Riedhammer KM, Seitz B, Robin NH, Goodloe D, Mignot C, Keren B, Cox H, Jarvis J, Hempel M, Gibson CF, Tran Mau-Them F, Vitobello A, Bruel AL, Sorlin A, Mehta S, Raymond FL, Gilmore K, Powell BC, Weck K, Li C, Vulto-van Silfhout AT, Giacomini T, Mancardi MM, Accogli A, Salpietro V, Zara F, Vora NL, Davis EE, Burdine R, Bhoj E. Abrogation of MAP4K4 protein function causes congenital anomalies in humans and zebrafish. Sci Adv. 2023 Apr 28;9(17):eade0631. doi: 10.1126/sciadv.ade0631. This is an open-access article distributed under the terms of the Creative Commons Attribution-NonCommercial license, which permits use, distribution, and reproduction in any medium, so long as the resultant use is not for commercial advantage and provided the original work is properly cited.

Recommended Citation

Patterson, Victoria; Grange, Dorothy K; and et al., "Abrogation of MAP4K4 protein function causes congenital anomalies in humans and zebrafish." Science advances. 9, 17. eade0631 (2023).
https://digitalcommons.wustl.edu/oa_4/2019

Department

ICTS (Institute of Clinical and Translational Sciences)

Additional Links

Supplemental material is available for this article at publisher site.