HGF/c-MET pathway inhibition combined with chemotherapy increases cytotoxic T-cell infiltration and inhibits pancreatic tumour growth and metastasis

Authors

Alpha Raj Mekapogu, University of New South Wales
Zhihong Xu, University of New South Wales
Srinivasa Pothula, University of New South Wales
Chamini Perera, University of New South Wales
Tony Pang, University of New South Wales
S M Zahid Hosen, University of New South Wales
Vishnu Damalanka, Washington University School of Medicine in St. Louis
James Janetka, Washington University School of Medicine in St. Louis
David Goldstein, University of New South Wales
Romano Pirola, University of New South Wales
Jeremy Wilson, University of New South Wales
Minoti Apte, University of New South Wales

Journal

Cancer Letters

Publication Date

8-1-2023

Volume

568

First Page

216286

Document Type

Open Access Publication

DOI

10.1016/j.canlet.2023.216286

Rights and Permissions

Mekapogu AR, Xu Z, Pothula S, Perera C, Pang T, Hosen SMZ, Damalanka V, Janetka J, Goldstein D, Pirola R, Wilson J, Apte M. HGF/c-Met pathway inhibition combined with chemotherapy increases cytotoxic T-cell infiltration and inhibits pancreatic tumour growth and metastasis. Cancer Lett. 2023 Aug 1;568:216286. doi: 10.1016/j.canlet.2023.216286. © 2023 The Authors. Published by Elsevier B.V. This is an open access article under the CC BY license (http://creativecommons.org/licenses/by/4.0/).

Recommended Citation

Mekapogu, Alpha Raj; Xu, Zhihong; Pothula, Srinivasa; Perera, Chamini; Pang, Tony; Hosen, S M Zahid; Damalanka, Vishnu; Janetka, James; Goldstein, David; Pirola, Romano; Wilson, Jeremy; and Apte, Minoti, "HGF/c-MET pathway inhibition combined with chemotherapy increases cytotoxic T-cell infiltration and inhibits pancreatic tumour growth and metastasis." Cancer Letters. 568, 216286 (2023).
https://digitalcommons.wustl.edu/oa_4/2464

Department

ICTS (Institute of Clinical and Translational Sciences)

Additional Links

Supplemental material is available for this article at publisher site.