PPTC7 maintains mitochondrial protein content by suppressing receptor-mediated mitophagy

Authors

Natalie M Niemi, Washington University School of Medicine in St. Louis
Lianjie Wei, Washington University School of Medicine in St. Louis
Andrew J Smith, Washington University School of Medicine in St. Louis
Merima Forny, Washington University School of Medicine in St. Louis
David J Pagliarini, Washington University School of Medicine in St. Louis
et al.

Journal

Nature Communications

Publication Date

10-13-2023

Volume

14

Issue

1

First Page

6431

Document Type

Open Access Publication

DOI

10.1038/s41467-023-42069-w

Rights and Permissions

Niemi, N.M., Serrano, L.R., Muehlbauer, L.K. et al. PPTC7 maintains mitochondrial protein content by suppressing receptor-mediated mitophagy. Nat Commun 14, 6431 (2023). https://doi.org/10.1038/s41467-023-42069-w This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

Recommended Citation

Niemi, Natalie M; Wei, Lianjie; Smith, Andrew J; Forny, Merima; Pagliarini, David J; and et al., "PPTC7 maintains mitochondrial protein content by suppressing receptor-mediated mitophagy." Nature Communications. 14, 1. 6431 (2023).
https://digitalcommons.wustl.edu/oa_4/3072

Department

ICTS (Institute of Clinical and Translational Sciences)

Additional Links

Supplemental material is available for this article at publisher site.