Implications of oligomeric amyloid-beta (oAβ42) signaling through α7β2-nicotinic acetylcholine receptors (nAChRs) on basal forebrain cholinergic neuronal intrinsic excitability and cognitive decline

Authors

Andrew A George, St. Joseph's Hospital and Medical Center
Jaime M Vieira, Arizona State University at the Tempe Campus
Cameron Xavier-Jackson, University of Bath
Michael T Gee, University of Arizona
John R Cirrito, Washington University School of Medicine in St. Louis
Heather A Bimonte-Nelson, Arizona State University at the Tempe Campus
Marina R Picciotto, Yale University
Ronald J Lukas, St. Joseph's Hospital and Medical Center
Paul Whiteaker, St. Joseph's Hospital and Medical Center

Journal

The Journal of neuroscience

Publication Date

1-20-2021

Volume

41

Issue

3

First Page

555

Last Page

575

Document Type

Open Access Publication

DOI

10.1523/JNEUROSCI.0876-20.2020

Rights and Permissions

George AA, Vieira JM, Xavier-Jackson C, Gee MT, Cirrito JR, Bimonte-Nelson HA, Picciotto MR, Lukas RJ, Whiteaker P. Implications of Oligomeric Amyloid-Beta (oAβ42) Signaling through α7β2-Nicotinic Acetylcholine Receptors (nAChRs) on Basal Forebrain Cholinergic Neuronal Intrinsic Excitability and Cognitive Decline. J Neurosci. 2021 Jan 20;41(3):555-575. doi: 10.1523/JNEUROSCI.0876-20.2020. Copyright © 2021 the authors.

Recommended Citation

George, Andrew A; Vieira, Jaime M; Xavier-Jackson, Cameron; Gee, Michael T; Cirrito, John R; Bimonte-Nelson, Heather A; Picciotto, Marina R; Lukas, Ronald J; and Whiteaker, Paul, "Implications of oligomeric amyloid-beta (oAβ42) signaling through α7β2-nicotinic acetylcholine receptors (nAChRs) on basal forebrain cholinergic neuronal intrinsic excitability and cognitive decline." The Journal of neuroscience. 41, 3. 555 - 575. (2021).
https://digitalcommons.wustl.edu/oa_4/3118

Department

ICTS (Institute of Clinical and Translational Sciences)