Homotopic contralesional excitation suppresses spontaneous circuit repair and global network reconnections following ischemic stroke

Authors

Annie R Bice, Washington University School of Medicine in St. Louis
Qingli Xiao, Washington University School of Medicine in St. Louis
Justin Kong, Washington University in St. Louis
Ping Yan, Washington University School of Medicine in St. Louis
Zachary Pollack Rosenthal, Washington University School of Medicine in St. Louis
Andrew W Kraft, Washington University School of Medicine in St. Louis
Karen P Smith, Washington University School of Medicine in St. Louis
Tadeusz Wieloch, Lund University
Jin-Moo Lee, Washington University School of Medicine in St. Louis
Joseph P Culver, Washington University School of Medicine in St. Louis
Adam Q Bauer, Washington University School of Medicine in St. Louis

Journal

Elife

Publication Date

6-20-2022

Volume

11

First Page

e68852

Document Type

Open Access Publication

DOI

10.7554/eLife.68852

Rights and Permissions

Bice AR, Xiao Q, Kong J, Yan P, Rosenthal ZP, Kraft AW, Smith KP, Wieloch T, Lee JM, Culver JP, Bauer AQ. Homotopic contralesional excitation suppresses spontaneous circuit repair and global network reconnections following ischemic stroke. Elife. 2022 Jun 20;11:e68852. doi: 10.7554/eLife.68852. Copyright Bice et al. This article is distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use and redistribution provided that the original author and source are credited.

Recommended Citation

Bice, Annie R; Xiao, Qingli; Kong, Justin; Yan, Ping; Rosenthal, Zachary Pollack; Kraft, Andrew W; Smith, Karen P; Wieloch, Tadeusz; Lee, Jin-Moo; Culver, Joseph P; and Bauer, Adam Q, "Homotopic contralesional excitation suppresses spontaneous circuit repair and global network reconnections following ischemic stroke." Elife. 11, e68852 (2022).
https://digitalcommons.wustl.edu/oa_4/357