HIV-1 capsid stability and reverse transcription are finely balanced to minimize sensing of reverse transcription products via the cGAS-STING pathway

Authors

Jenna E Eschbach, Washington University School of Medicine in St. Louis
Maritza Puray-Chavez, Washington University School of Medicine in St. Louis
Shawn Mohammed, Washington University School of Medicine in St. Louis
Qiankun Wang, Washington University School of Medicine in St. Louis
Ming Xia, Washington University School of Medicine in St. Louis
Lin-Chen Huang, Washington University School of Medicine in St. Louis
Liang Shan, Washington University School of Medicine in St. Louis
Sebla B Kutluay, Washington University School of Medicine in St. Louis

Journal

mBio

Publication Date

5-8-2024

Volume

15

Issue

5

First Page

e0034824

Document Type

Open Access Publication

DOI

10.1128/mbio.00348-24

Rights and Permissions

Eschbach JE, Puray-Chavez M, Mohammed S, Wang Q, Xia M, Huang L-C, Shan L, Kutluay SB. HIV-1 capsid stability and reverse transcription are finely balanced to minimize sensing of reverse transcription products via the cGAS-STING pathway. mBio. 2024 May 8;15(5):e0034824. doi: 10.1128/mbio.00348-24. © 2024 Eschbach et al. This is an open-access article distributed under the terms of the Creative Commons Attribution 4.0 International license.

Recommended Citation

Eschbach, Jenna E; Puray-Chavez, Maritza; Mohammed, Shawn; Wang, Qiankun; Xia, Ming; Huang, Lin-Chen; Shan, Liang; and Kutluay, Sebla B, "HIV-1 capsid stability and reverse transcription are finely balanced to minimize sensing of reverse transcription products via the cGAS-STING pathway." mBio. 15, 5. e0034824 (2024).
https://digitalcommons.wustl.edu/oa_4/3743

Department

ICTS (Institute of Clinical and Translational Sciences)

Additional Links

Supplemental material is available for this article at publisher site.