Profilin-1 regulates DNA replication forks in a context-dependent fashion by interacting with SNF2H and BOD1L

Authors

Cuige Zhu, Washington University School of Medicine in St. Louis
Mari Iwase, Washington University School of Medicine in St. Louis
Ziqian Li, Washington University School of Medicine in St. Louis
Faliang Wang, Washington University School of Medicine in St. Louis
Annabel Quinet, Washington University School of Medicine in St. Louis
Alessandro Vindigni, Washington University School of Medicine in St. Louis
Jieya Shao, Washington University School of Medicine in St. Louis

Journal

Nature Communications

Publication Date

11-1-2022

Volume

13

Issue

1

First Page

6531

Document Type

Open Access Publication

DOI

10.1038/s41467-022-34310-9

Rights and Permissions

Zhu, C., Iwase, M., Li, Z. et al. Profilin-1 regulates DNA replication forks in a context-dependent fashion by interacting with SNF2H and BOD1L. Nat Commun 13, 6531 (2022). https://doi.org/10.1038/s41467-022-34310-9 This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

Recommended Citation

Zhu, Cuige; Iwase, Mari; Li, Ziqian; Wang, Faliang; Quinet, Annabel; Vindigni, Alessandro; and Shao, Jieya, "Profilin-1 regulates DNA replication forks in a context-dependent fashion by interacting with SNF2H and BOD1L." Nature Communications. 13, 1. 6531 (2022).
https://digitalcommons.wustl.edu/oa_4/882

Additional Links

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