Journal
Clinical Epigenetics
Publication Date
5-10-2024
Volume
16
Issue
1
First Page
64
Document Type
Open Access Publication
DOI
10.1186/s13148-024-01676-0
Rights and Permissions
Bloks, N.G.C., Dicks, A., Harissa, Z. et al. Hyper-physiologic mechanical cues, as an osteoarthritis disease-relevant environmental perturbation, cause a critical shift in set points of methylation at transcriptionally active CpG sites in neo-cartilage organoids. Clin Epigenet 16, 64 (2024). https://doi.org/10.1186/s13148-024-01676-0 This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.
Recommended Citation
Bloks, Niek G C; Dicks, Amanda; Harissa, Zainab; Nelissen, Rob G H H; Hajmousa, Ghazaleh; Ramos, Yolande F M; de Almeida, Rodrigo Coutinho; Guilak, Farshid; and Meulenbelt, Ingrid, "Hyper-physiologic mechanical cues, as an osteoarthritis disease-relevant environmental perturbation, cause a critical shift in set points of methylation at transcriptionally active CpG sites in neo-cartilage organoids." Clinical Epigenetics. 16, 1. 64 (2024).
https://digitalcommons.wustl.edu/oa_4/5811
Department
ICTS (Institute of Clinical and Translational Sciences)
Additional Links
Supplemental material is available for this article at publisher site.
