ISG15 negatively regulates RIPK3-mediated cell death and viral pathogenesis

Authors

• Yi-Chieh Perng, Washington University School of Medicine in St. Louis
• Jessica M C Warsaw, Washington University School of Medicine in St. Louis
• Sachendra S Bais, Washington University School of Medicine in St. Louis
• Bradley E Hiller, Washington University School of Medicine in St. Louis
• Marissa C Locke, Washington University School of Medicine in St. Louis
• David J Morales-Heil, Washington University School of Medicine in St. Louis
• Chaoqun Li, University of Pittsburgh
• Alissa R Young, Washington University School of Medicine in St. Louis
• Kristen J Monte, Washington University School of Medicine in St. Louis
• Robert E Schmidt, Washington University School of Medicine in St. Louis
• Douglas R Green, St. Jude Children's Research Hospital
• Yi-Nan Gong, University of Pittsburgh
• Deborah J Lenschow, Washington University School of Medicine in St. Louis

Journal

Cell Reports

Publication Date

5-26-2026

Volume

45

Issue

5

First Page

117360

Document Type

Open Access Publication

DOI

10.1016/j.celrep.2026.117360

Rights and Permissions

Perng YC, Warsaw JMC, Bais SS, Hiller BE, Locke MC, Morales-Heil DJ, Li C, Young AR, Monte KJ, Schmidt RE, Green DR, Gong YN, Lenschow DJ. ISG15 negatively regulates RIPK3-mediated cell death and viral pathogenesis. Cell Rep. 2026 May 26;45(5):117360. doi: 10.1016/j.celrep.2026.117360. © 2026 The Authors. Published by Elsevier Inc. This is an open access article under the CC BY-NC license (http://creativecommons.org/licenses/by-nc/4.0/)

Recommended Citation

Perng, Yi-Chieh; Warsaw, Jessica M C; Bais, Sachendra S; Hiller, Bradley E; Locke, Marissa C; Morales-Heil, David J; Li, Chaoqun; Young, Alissa R; Monte, Kristen J; Schmidt, Robert E; Green, Douglas R; Gong, Yi-Nan; and Lenschow, Deborah J, "ISG15 negatively regulates RIPK3-mediated cell death and viral pathogenesis." Cell Reports. 45, 5. 117360 (2026).
https://digitalcommons.wustl.edu/oa_4/7293

Department

ICTS (Institute of Clinical and Translational Sciences)

Additional Links

Supplemental material is available for this article at publisher site.